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dc.contributor.author Peña-Oyarzún, Daniel
dc.contributor.author San Martin, Carla
dc.contributor.author Hernández-Cáceres, María Paz
dc.contributor.author Lavandero, Sergio
dc.contributor.author Morselli, Eugenia
dc.contributor.author Budini, Mauricio
dc.contributor.author Burgos, Patricia V.
dc.contributor.author Criollo, Alfredo
dc.date.accessioned 2024-09-26T00:29:54Z
dc.date.available 2024-09-26T00:29:54Z
dc.date.issued 2022-08-05
dc.identifier.issn 1664-2392
dc.identifier.uri https://repositorio.uss.cl/handle/uss/12357
dc.description Publisher Copyright: Copyright © 2022 Peña-Oyarzún, San Martin, Hernández-Cáceres, Lavandero, Morselli, Budini, Burgos and Criollo.
dc.description.abstract Autophagy is an intracellular degradation mechanism that allows recycling of organelles and macromolecules. Autophagic function increases metabolite availability modulating metabolic pathways, differentiation and cell survival. The oral environment is composed of several structures, including mineralized and soft tissues, which are formed by complex interactions between epithelial and mesenchymal cells. With aging, increased prevalence of oral diseases such as periodontitis, oral cancer and periapical lesions are observed in humans. These aging-related oral diseases are chronic conditions that alter the epithelial-mesenchymal homeostasis, disrupting the oral tissue architecture affecting the quality of life of the patients. Given that autophagy levels are reduced with age, the purpose of this review is to discuss the link between autophagy and age-related oral diseases. en
dc.language.iso eng
dc.relation.ispartof vol. 13 Issue: Pages:
dc.source Frontiers in Endocrinology
dc.title Autophagy in aging-related oral diseases en
dc.type Artículo de revisión
dc.identifier.doi 10.3389/fendo.2022.903836
dc.publisher.department Facultad de Medicina y Ciencia


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